Hydrocephalus.- How to Select Patients with Normal Pressure Hydrocephalus for Shunting?.- Agreement Between CSF Flow Dynamic in MRI and ICP Monitoring in the Diagnosis of Normal Pressure Hydrocephalus. Sensitivity and Specificity of CSF Dynamics to Predict Outcome.- Shunting to the Sagittal Sinus.- Why Valve Opening Pressure Plays a Relatively Minor Role in the Postural ICP Response to Ventricular Shunts in Normal Pressure Hydrocephalus: Modeling and Implications.- Dynamic Shunt Testing Applying Short Lasting Pressure Waves — Inertia of Shunt Systems.- Factors Determining Mean ICP in Hydrocephalic Patients with Hakim-Programmable Valve: Implications of the Parallel Arrangement of the CSF Outflow Resistance and Shunt.- Shunt Testing in-Vivo: A Method Based on the Data from the UK Shunt Evaluation Laboratory.- Compensatory Mechanisms in Patients with Benign Intracranial Hypertension Syndrome.- Intracranial Hypertension after Surgery in Patients with Chiari I Malformation and Normal or Moderate Increase in Ventricular Size.- The Role of Cerebral Blood Flow and Cerebrovascular Reserve Capacity in the Diagnosis of Chronic Hydrocephalus — A PET-Study on 60 Patients.- PET-Studies in Idiopathic Chronic Hydrocephalus Before and after Shunt-Treatment: The Role of Risk Factors for Cerebrovascular Disease (CVD) on Cerebral Hemodynamics.- Regional Cerebral Blood Flow Profiles of Shunt-Responder in Idiopathic Chronic Hydrocephalus — A 15-O-Water PET-Study.- Neuropsychological Testing to Improve Surgical Management of Patients with Idiopathic Chronic Hydrocephalus after Shunt Treatment.- Neuropsychological Sequels to Changes in Global Cerebral Blood Flow and Cerebrovascular Reserve Capacity after Shunt Treatment in Chronic Hydrocephalus — A Quantitative PET-Study.- Communicating Hydrocephalus: The Biomechanics of Progressive Ventricular Enlargement Revisited.- Clinical Management of Head Injury.- Is CPP Therapy Beneficial for all Patients with High ICP?.- Metabolic Suppressive Therapy as a Treatment for Intracranial Hypertension — Why it Works and when it Fails.- Can Hyperventilation Improve Cerebral Microcirculation in Patients with High ICP?.- Cerebral Perfusion Pressure and SPECT in Patients after Craniocerebral Injury with Transtentorial Herniation.- Decompressive Craniectomy Following Traumatic Brain Injury: ICP, CPP and Neurological Outcome.- Evolving Lesions in Traumatic Subarachnoid Hemorrhage: Prospective Study of 110 Patients with Emphasis on the Role of ICP Monitoring.- Effect of Early Induction of Hypothermia on Severe Head Injury.- Significance of Temperature Difference Between Cerebral Cortex and Axilla in Patients Under Hypothermia Management.- ICP is Lower during Propofol Anaesthesia Compared to Isoflurane and Sevoflurane.- Effects of Positive End-Expiratory Pressure on Intracranial Pressure and Cerebral Perfusion Pressure.- The Safety of the Open Lung Approach in Neurosurgical Patients.- Optimizing Cerebral Perfusion Pressure (CPP) During Fiberoptic Bronchoscopy in Severe Head Injury: Effect of Hyperventilation.- Assessment of Cerebrovascular Reactivity.- ICP and CBF Regulation: Effect of the Decompressive Craniectomy.- ICP and CBF Regulation: A new Hypothesis to Explain the “Windkessel” Phenomenon.- Clinical Significance of Cerebral Autoregulation.- Routine Utilization of the Transient Hyperaemic Response Test after Aneurysmal Subarachnoid Haemorrhage.- Is Transcranial Doppler Ultrasonography (TCD) Good Enough in Determining CO2 Reactivity and Pressure Autoregulation in Head-Injured Patients?.- Comparison Between two Static Autoregulation Evaluation Methods.- Asymmetry of Cerebral Autoregulation Following Head Injury.- Oscillations in the Near-Infrared Signal in Patients with Severe Head Injury.- Slow Rhythmic Intracranial CSF and Blood Flow: Registered by MRI.- Cyclic Variation of Cerebral Pial Arteriolar Diameter Synchronized with Positive Pressure Inhalation.- Spectral Characteristics of B-Waves and Other low-Frequency Activity.- Variation of Proposed Correlation Indices of Cerebrovascular Reactivity with Change of Arteriolar Diameter.- Use of Resistance-Area Product Derived from Doppler MCA Velocity to Estimate the Range of Active Cerebrovascular Regulation.- ICP Analysis.- Online Analysis of Intracranial Pressure Waves.- Information Technology and CPP Management in Neuro Intensive Care.- Multi-Centre Assessment of the Spiegelberg Compliance Monitor: Interim Results.- Clinical Experience in the use of the Spiegelberg Automated Compliance Device in the Assessment of Patients with Hydrocephalus.- Continuous Cerebral Compliance Monitoring in Severe Head Injury: Its Relationship with Intracranial Pressure and Cerebral Perfusion Pressure.- Intracranial Compliance as Bed-Side Monitoring Technique in Severely Head-Injured Patients.- An Experimental Evaluation of a new Intraparenchymal Continuous Compliance Probe: Preliminary Studies.- An Estimated Compliance Index Derived from Intracranial Pressure Recording.- Non-Invasive Assessment of Intracranial Compliance Using a New 2-D “Transit Time” vs “Frequency Centroid” Map.- Experimental: Physiology, HI Models, SAH, Ischaemia, Hydrocephalus.- Approximate Entropy: A Regularity Statistic for Assessment of Intracranial Pressure.- Simultaneous Measurement of Local Cortical Blood Flow and Tissue Oxygen Saturation by Near Infra-Red Laser Doppler Flowmetry and Remission Spectroscopy in the Pig Brain.- Cerebral Veno-Arterial pCO2 Difference as an Estimator of Uncompensated Cerebral Hypoperfusion.- Glial and Neuronal Serum Markers After Controlled Cortical Impact Injury in the Rat.- Long-Time in-Vivo Metabolic Monitoring Following Experimental Brain Contusion Using Proton Magnetic Resonance Spectroscopy.- Determination of Contusion and Oedema Volume by MRI Corresponds to Changes of Brain Water Content Following Controlled Cortical Impact Injury.- Secondary Insults Worsen Blood Brain Barrier Dysfunction Assessed by MRI in Cerebral Contusion.- Neuronal Activity Determined by Quantitative EEG and Cortical Microdialysis is Increased Following Controlled Cortical Impact Injury in Rats.- Norepinephrine is Superior to Dopamine in Increasing Cortical Perfusion Following Controlled Cortical Impact Injury in Rats.- Cortical Hypoperfusion Precedes Hyperperfusion Following Controlled Cortical Impact Injury in Rats.- Effects of Brain Hypothermia on Brain Edema Formation after Intracerebral Hemorrhage in Rats.- Appropriate Cerebral Perfusion Pressure During Rewarming after Therapeutic Hypothermia.- Diffuse Brain Injury Complicated by Acute Subdural Hematoma and Secondary Insults in the Rodents: The Effect of Surgical Evacuation.- Diffuse Brain Injury Complicated by Acute Subdural Hematoma in The Rodents: The Effect of Early or Delayed Surgical Evacuation.- Traumatic Subarachnoidal Hemorrhage in the Developing Rat.- Experimental Intracerebral Hemorrhage: Effect of Lysed Erythrocytes on Brain Edema and Blood-Brain Barrier Permeability.- Brain Edema after Intracerebral Hemorrhage: The Effects of Systemic Complement Depletion.- Metabolic Alterations in Cerebrospinal Fluid from a Double Hemorrhage Model of Dogs.- Effect of Ischemic Preconditioning on Edema Formation and Cerebrovascular Injury Following Focal Cerebral Ischemia.- ICP Monitoring Following Bilateral Carotid Occlusion in GFAP-Null Mice.- Spinal Cerebrospinal Fluid Pathways and Their Significance for the Compensation of Kaolin- Hydrocephalus.- Temporal and Regional Profile of Neuronal and Glial Cellular Injury after Induction of Kaolin Hydrocephalus.- Brain Biochemical Monitoring and Imaging.- Mechanisms of the Mass Effect of Cerebral Contusion: ICP Monitoring and Diffusion MRI Study.- Incidence of Intracranial Hypertension Related to Jugular Bulb Oxygen Saturation Disturbances in Severe Traumatic Brain Injury Patients.- Detection of Early Ischemia in Severe Head Injury by Means of Arteriovenous Lactate Differences and Jugular Bulb Oxygen Saturation. Relationship with CPP, Severity Indexes and Outcome. Prelimiary Analysis.- Non Invasive Local Cerebral Oxygenation Monitoring Using a Combination of Light and Ultrasound.- Brain Tissue PO2 Correlation with Cerebral Blood Flow.- Coexistence of Regional Cerebral Hypoxia with Normal or Hyperemic Brain Detected by Global Monitoring Methods. Analysis of Apparently Contradictory Findings Based on the Siggaard-Andersen Model of Tissue Hypoxia.- Therapeutic Aspects of Brain Tissue p02 Monitoring after Subarachnoid Hemorrhage.- Dynamical Correlation Between Tissue pO2 and near Infrared Spectroscopy.- Effects of Hyperoxia on Brain Tissue Oxygen Tension in Cerebral Focal Lesions.- Detection of Secondary Insults by Brain Tissue pO2 and Bedside Microdialysis in Severely Head-Injury.- Intraoperative Monitoring of Brain Tissue Oxygen and Carbon Dioxide Pressure in Peritumoural Oedema by Stereotactic Placement of Multiparameter Microsensors.- Effects of Variation in Cerebral Haemodynamics During Aneurysm Surgery on Brain Tissue Oxygen and Metabolism.- Microdialysate Nitrate/Nitrite Levels Following Severe Head Injury.- Neurochemical Changes in Ventilated Head-Injured Patients with Cerebral Perfusion Pressure Treatment Failure.- Satellite-Neurochemical Monitoring.- Third International Satellite Conference on Neuro-Chemical Monitoring (Satellite Panel Discussion).- Measurement of the Nitric Oxide Metabolites Nitrate and Nitrite in the Human Brain by Microdialysis.- Comparison of Microdialysate Arginine and Glutamate Levels in Severely Head-Injured Patients.- Temporal Profiles of Extracellular Nitric Oxide Metabolites Following Aneurysmal Subarachnoid Hemorrhage.- Delayed Increase in Extracellular Glycerol with Post-Traumatic Electrographic Epileptic Activity: Support for the Theory that Seizures Induce Secondary Injury.- Clinical Cerebral Microdialysis — Determining the True Extracellular Concentration.- Extracellular Lactate/Pyruvate and Glutamate Changes in Patients during Peroperative Episodes of Cerebral Ischaemia.- The Evaluation of Brain CBF and Mitochondrial Function by a Fiber Optic Tissue Spectroscope in Neurosurgical Patients.- Application of Chemical Shift Imaging for Measurement of NAA in Head Injured Patients.- Investigation of Extracellular Amino Acid Release in Children with Severe Head Injury Using Microdialysis. A Pilot Study.