Published July, 2015
By Nathan A. Berger
Publisher: Springer (Springer International Publishing AG)
Series: Energy Balance and Cancer
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Availability: Available

This volume provides a transdisciplinary and translational review of many of the leading murine models used to study the mechanisms, mediators and biomarkers linking energy balance to cancer.

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Published June, 2015
By Nathan A. Berger and Andrew J. Dannenberg
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Energy Balance and Cancer
Rating:
Availability: Available

In addition to its metabolic and endocrinologic effects, obesity and adipose tissue have now been shown to be associated with low grade inflammation resulting in cellular and humoral inflammatory factors of which the latter may act by endocrine, paracrine and autocrine mechanisms.

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Published November, 2014
By Nathan A. Berger, Karen Steindorf and Cornelia M. Ulrich
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Energy Balance and Cancer
Rating:
Availability: Available

This book examines the effect of exercise in biological pathways in tumor development. It explores the impact of exercise and ROS, RNOS and prostaglandin on tumor growth, and examines the relationship between physical activity and cancer.

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Published August, 2014
By Nathan A. Berger, Deborah J. Bowen and Gerald V. Denis
Publisher: Springer (Springer International Publishing AG)
Series: Energy Balance and Cancer
Rating:
Availability: Available

This volume reviews disparities in cancer genetics, etiology, treatment and survivorship that are associated with differences in energy balance and how those differences and disparities may be affected by geography, socioeconomic status, ethnicity, biology, behavior and others.

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Published April, 2014
By Nathan A. Berger and Steven D Mittelman
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Energy Balance and Cancer
Rating:
Availability: Available

The obesity pandemic continues to increase on a world-wide basis with over 70% of the United States population being either overweight or obese. Another important aspect of the association of obesity with hematologic malignancies is the increased incidence of obesity in children who successfully complete therapy for acute lymphoblastic leukemia.

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Published April, 2014
By Nathan A. Berger and Sanford D. Markowitz
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Energy Balance and Cancer
Rating:
Availability: Available

The gastrointestinal track provides one of the distinct systems where multiple malignancies, including adenocarcinoma of the pancreas, esophagus and colon are each associated with obesity.

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Published December, 2013
By Nathan A. Berger and Susan Redline
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Energy Balance and Cancer
Rating:
Availability: Available

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Published May, 2013
By Nathan A. Berger, Andrew Dannenberg and Andrew J. Dannenberg
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Energy Balance and Cancer
Rating:
Availability: Available

In addition to its metabolic and endocrinologic effects, obesity and adipose tissue have now been shown to be associated with low grade inflammation resulting in cellular and humoral inflammatory factors of which the latter may act by endocrine, paracrine and autocrine mechanisms.

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Published April, 2013
By I. George Fantus
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Energy Balance and Cancer
Rating:
Availability: Available

This book reviews the epidemiological associations between insulin resistance and cancer. For example some tumors present increased quantities of the fetal form of the insulin receptor, unique regulation of oxidative (Krebs' cycle) metabolism (Warburg effect), as well as mutations in various relevant signaling pathways.

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Published December, 2012
By Anne McTiernan
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Energy Balance and Cancer
Rating:
Availability: Available

The World Health Organization estimates that 25 percent of common cancers can be prevented through regular physical activity and weight control. There are several plausible mechanisms linking lack of physical activity and increased adiposity to cancer risk, supported by results from animal experiments and human intervention studies.

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