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Showing 7 of 7 results for "Series: Springer Series in Translational Stroke Research"
 
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Published December, 2014
By John H. Zhang and Li-Ru Zhao
Publisher: Springer (Springer International Publishing AG)
Series: Springer Series in Translational Stroke Research
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Cellular therapy for stroke and neural trauma has gained worldwide attention during the last decade and has shown some promising results. Various cells, including neural stem cells, bone marrow stem cells, endothelial progenitor cells, and many others have had protective or regenerative effects in animal models.

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White matter injury can result from both ischemic and hemorrhagic stroke as well as a host of other CNS diseases and conditions such as neonatal injuries, neurodegenerative disorders including Alzheimer's disease, traumatic brain injuries, carbon monoxide poisoning, and drug or alcohol overdoses.

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Published November, 2013
By John H. Zhang, Jun Chen, Xiaoming Hu and Mary Stenzel-Poore
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Springer Series in Translational Stroke Research
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Recent research has revealed the importance of immunological mechanisms and inflammation in delaying damage and/or promoting repair after an acute injury to the central nervous system.

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While there are a number of books on the market that deal with neuronal mechanisms and targets, the proposed book will be the only one to cover the vascular aspects of CNS trauma.

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Published September, 2012
By John H. Zhang, Jeffrey M. Gidday and Miguel A. Perez-Pinzon
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Springer Series in Translational Stroke Research
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Cerebral preconditioning is a phenomenon wherein a mild insult or stress induces cellular and tissue adaptation or tolerance to a later, severe injury, therefore reflecting the efficacy of endogenous mechanisms of cerebrovascular protection.

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Published May, 2012
By John H. Zhang and Yang V. Li
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Springer Series in Translational Stroke Research
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Cellular events that occur following stroke include the excessive releases of excitatory amino acids, alterations in the genomic responses, mitochondrial injury producing reactive oxygen and nitrogen species (ROS), and secondary injury, often in the setting of reperfusion.

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Published March, 2012
By John H. Zhang and Paul A. Lapchak
Publisher: Springer (Springer-Verlag New York Inc.)
Series: Springer Series in Translational Stroke Research
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This volume sets a basis for effective translational research. Authored by experts in the field of translational stroke research, each chapter specifically addresses one or more components of preclinical stroke research.

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